Deregulation of Autophagy
Autophagic Vacuoles
Lysosomes
Phosphorylated
Phosphorylation
Calcium Channels
AMPK
Prion
Maleria
Corallicolid
Linking Cerebral Malaria Pathogenesis to APOE-Mediated Amyloidosis: Observations and Hypothesis
https://link.springer.com/article/10.1007/s12035-024-04366-3
Possible roles of amyloids in malaria pathophysiology
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5137859/
Lysosomal calcium signalling regulates autophagy through calcineurin and TFEB
https://www.nature.com/articles/ncb3114
Ca2+ signals critical for egress and gametogenesis in malaria parasites depend on a multipass membrane protein that interacts with PKG
https://www.science.org/doi/10.1126/sciadv.abe5396
Evidence for glycoprotein transport into complex plastids
https://www.pnas.org/doi/10.1073/pnas.1301945110
Mycoplasma invasion into host cells: An integrated model of infection strategy
https://onlinelibrary.wiley.com/doi/10.1111/mmi.15232
Mycoplasma fermentans
Binds to and Invades HeLa Cells: Involvement of Plasminogen and Urokinase
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC517474/
Regulation of calcium homeostasis and flux between the endoplasmic reticulum and the cytosol
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9218512/
Protein Folding in the Endoplasmic Reticulum
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3632058/
Thiamine Repletion, Calcium Management, and the Heart
https://hormonesmatter.com/thiamine-repletion-calcium-management-heart/
Protein Targeting to the Malaria Parasite Plastid
https://onlinelibrary.wiley.com/doi/10.1111/j.1600-0854.2007.00660.x
..
HIF (1a) Protein
Hypoxia-Inducible Factor
1 Alpha
Prion
Calcium
..
Tin disrupts Calcium protein folding & stability.
The presence of calcium is relevant for both folding and stability of many protein domains
Organiotin compound in antifouling coating material of boat and ship could result in sex distortion of marine organism,as well as disturbing their calcium metabolism
Evidence indicates that calcium homeostasis in the brain is sensible to Triethyltin (TET) exposure, as it is also capable of inducing noradrenaline spontaneous release in rat cultured hippocampal slices by altering the functioning of calcium channels
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