Alanine
Glycine
Glutamine
Glutamate
Excitotoxicity
NMDA Overactivation
BMAA
B-Methylamino-L-Alanine
Diaminobutyric Acid (DAB)
Sulfate-Reducing Bacteria (SRB)
Methyl Group (CH3)
Methylation
Oxidation
Cyanobacteria
Microalgae
Plant Root Symbiont
GABA
DHEA
Pregnenolone
Butyrate Butter
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The Truth About Magnesium Glycinate and Breakouts
https://en.m.wikipedia.org/wiki/Excitotoxicity
https://en.m.wikipedia.org/wiki/NMDA_receptor_antagonist
https://www.mdpi.com/2073-4409/9/3/698
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Butyrate oxidation is the process where butyrate, a short-chain fatty acid, is metabolized by cells.
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Microbial BMAA and the Pathway for Parkinson’s Disease Neurodegeneration
https://pmc.ncbi.nlm.nih.gov/articles/PMC7019015/
Non-Proteinogenic Amino Acid β-N-Methylamino-L-Alanine (BMAA): Bioactivity and Ecological Significance
https://pmc.ncbi.nlm.nih.gov/articles/PMC9414260/
Oxidation of cyanobacterial neurotoxin beta-N-methylamino-L-alanine (BMAA) with chlorine, permanganate, ozone, hydrogen peroxide and hydroxyl radical
https://www.sciencedirect.com/science/article/abs/pii/S0043135418304342
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While BMAA itself is not a prion, it has been proposed that chronic exposure to BMAA might contribute to neurodegeneration by promoting protein misfolding and aggregation, a process resembling the prion-like mechanism observed in certain neurodegenerative diseases.
BMAA can be degraded through various oxidation methods, but the effectiveness can be influenced by factors like pH
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Cyanobacteria
Microalgae
Plant Root Symbiont
it was first discovered in Guam, from algae root symbiont that infects the berries?
is this the origin Prion discoved New Guinea?
match up the symptoms of BMAA with Long Covid, Malaria & HIV?
because if they put this microalgae in a mycoplasma &/or gut bacteria as a symbiont, it would behave exactly like what we are seeing.
RE: Intercellular Homeostasis