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mikewick77(49)
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Vitamin E
Alpha-Tocopherol

Farnesyl
Gamma-Tocopherol Methyltransferase (γ-TMT)
Tocopherol Biosynthesis

An Update on Vitamin E, Tocopherol and Tocotrienol

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6257355/

Vitamin E function and requirements in relation to PUFA

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4594047/

Tocopherols and Tocotrienols in Common and Emerging Dietary Sources: Occurrence, Applications, and Health Benefits

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5085773/

Methyltransferases: Functions and Applications

https://chemistry-europe.onlinelibrary.wiley.com/doi/10.1002/cbic.202200212

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"More than 60 years ago, a vitamin E deficiency was known to cause a brain disease, sometimes associated with sterility and muscular dystrophy. The symptoms of the brain disease were similar to those of “mad cow disease,” and the condition is now usually called “crazy chick disease.” Veterinarians are usually taught that it is caused by a selenium deficiency, but it is actually the result of an excess of PUFA in the diet, and is exacerbated by increased iron or other oxidants, and prevented by increased vitamin E, selenium, or substitution of saturated fats for the unsaturated."

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MTHFR mutation/variant
C677T & A1298C

CH3
Methylation
Methylene
Methionine
Methyltransferase
Methylcobalamin
Trimethylglycine
Methylfolate

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MTHFR Genes C677T vs A1298C

https://mthfrgenehealth.com/mthfr-genes-c677t-vs-a1298c/

MTHFR Gene Mutations Correlate with White Matter Disease Burden and Predict Cerebrovascular Disease and Dementia

Epigenetic Factors in Late-Onset Alzheimer’s Disease: MTHFR and CTH Gene Polymorphisms, Metabolic Transsulfuration and Methylation Pathways, and B Vitamins

C677T, A1298C, and A1793G polymorphisms on the age at onset, plasma homocysteine, and white matter lesions in Alzheimer's disease

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Hyperhomocysteinemia: Clinical Insights

https://journals.sagepub.com/doi/full/10.1177/1179573520962230

Homocysteine & MTHFR

https://mthfrgenehealth.com/homocysteine-mthfr/

Homocysteine Disfunction
Hyperhomocysteineaemia
Alcohol, Tobacco, Caffeine
pH Homeostasis

Excessive:
Alcohol, Coffee or Tea
Smoking
Lack of exercise
High salt intake
High fat diet (red meat)
High-fat (dairy)

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Levomefolic
Methylfolate

Medical foods for lowering homocysteine in hypertensive patients

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9832224/

KEY INGREDIENTS: L‐METHYLFOLATE, RIBOFLAVIN, AND NAC

Homocysteine Reduction

https://www.lifeextension.com/protocols/heart-circulatory/homocysteine-reduction

The Complete Guide to Folic Acid, Folate, and L-Methylfolate

https://www.seekinghealth.com/blogs/education/the-complete-guide-to-folic-acid-folate-and-l-methylfolate

Pteroylpolyglutamate

Pteroyl: polycyclic heterocyclic acyl radical

Acetate Revisited: A Key Biomolecule at the Nexus of Metabolism, Epigenetics and Oncogenesis—Part 1: Acetyl-CoA, Acetogenesis and Acyl-CoA Short-Chain Synthetases

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7689297/

What The Science Says About B3 Vitamins And Methylation

https://www.aboutnad.com/scientific-analysis/what-the-science-says-about-b3-vitamins-and-methylation

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Tocopherol (Vitamin E)
Trimethyltridecyl

Trimethylglycine (TMG)
Betaine Anhydrous

https://en.m.wikipedia.org/wiki/Betaine

https://en.m.wikipedia.org/wiki/Trimethylglycine

Betaine vs. TMG

https://thisvsthat.io/betaine-vs-tmg

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Homocysteine levels in schizophrenia and affective disorders—focus on cognition

Investigation of betaine as a novel psychotherapeutic for schizophrenia

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how exactly does half the population have MTHFR mutation/variant C677T & A1298C?

and why does this seem to be the major malfunction, in the B6 enzyme?

after learning about this, im again strongly suspicious of Candida being more of a slime mold symbiont of coral.

its more intelligent then justva fungus, aquires data & evolves, relocates around the body, and may be the true origin of MTHFR Polymorphism.

Candida ≈ Lyme

still dont think Candida is just a yeast, but a symbiont, meaning it can use yeast & mycoplasma as vectors.

its doing the same kind of thing, just slower, makes me think its generally the same critter.

Candida Cordyceps
Amoebae Amoebozoa

like a single unit fused together inside our neurological systems, manufacturing MTHFR Gene Mutation.

Candida Hyperparasite coinfection is the Cordyceps of mammals.. thats the general idea.

if a corpse is let to decompose, would this Prion grow into Candida Cordyceps spores?

seems like Candida behaves differently outside vs inside the body, Covid, Lyme, GWS, HIV are inserts into Candida, and therefore instant access to our neurological systems.

wonder if the MTHFR Homocysteine B6 enzyme lock has anything to do with Amyloidosis, Prion Hydrogel Peptides ect..

Amyloids and yeast prion biology

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7321840/

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MTHFR Gene
Mutation/Variant:
C677T & A1298C

Homocysteine Disfunction
Hyperhomocysteineaemia

Trimethylglycine (TMG)
Betaine Anhydrous

..

Yeast is a Beast – The MTHFR and Candida Connection

https://www.beyondmthfr.com/yeast-is-a-beast-the-mthfr-and-candida-connection/

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Hyperparasite
Endoparasitoid
Dictyostelid Pathogen
Cellular Slime Mold
Symbiont Endosymbiont

Candida Cordyceps
Amoebae Amoebozoa
Cavenderia Aureostipes
Physarum Polycephalum
Toxoplasma
Mycoplasma

https://en.m.wikipedia.org/wiki/Physarum_polycephalum

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https://en.m.wikipedia.org/wiki/Symbiodinium

https://en.m.wikipedia.org/wiki/Aiptasia

https://en.m.wikipedia.org/wiki/Myxozoa

https://en.m.wikipedia.org/wiki/Polypodium_hydriforme

@mikewick77: Vitamin E | Ecency