@mikewick77: Candidatus Candidate

if Spike is Prion, then nothing therapeutic or herbal will be sufficient, im talking about Thiamine as a very specific molecule structure to bind Prion, the only other things that will be as effective, are chemically identical to Thiamine, its because of the 5-Carbon Ring Scaffold Heterocyclic, with a Sulfur & Nitrogen.

B1 dismantling the Covid spike is the same idea for mycoplasma too, its so contagious that it must be mutated, meaning its not the mycoplasma itself, but what was put inside the mycoplasma.

cant prove this will actually happen, but looking at the pathways involved, match them up with things like mycoplasma incognitus & prion.

this B1 chemistry compound is too small for any parasite to hide, thats what is exciting about it, and its similar enough to an Alkaloid to actually wipe the parasitic data from its own toxic DNA, and flush out the mitochondria.

https://www.hormonesmatter.com/vitamin-therapy-paradox/

https://www.hormonesmatter.com/paradoxical-reactions-with-ttfd-the-glutathione-connection/

Mycoplasma fermentans infection induces human necrotic neuronal cell death via IFITM3-mediated amyloid-β (1–42) deposition

https://www.nature.com/articles/s41598-023-34105-y

Current Therapeutics, Their Problems, and Sulfur-Containing-Amino-Acid Metabolism as a Novel Target against Infections by “Amitochondriate” Protozoan Parasites

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1797636/

the connection im make by the combination of all this, its all matching up

Spike
Prion
Amyloid
Mycoplasma
Fermentans
Incognitus
Plastid
Plasmid
Protist mRNA

and if its correct, then by finding a cure to long covid (and vax) is the same cure to most other neurological diseases.

and the idea is the microbes are damn near indestructible, so they must be bound up & the Protist dna data must be completely wiped clean, and this may create a natrual immunity.

dont know how pathogens can form a defensive yet, because all the parasites have some kind of affinity to B1, and by flooding it will train them to leave it alone or die, reprogramming them.

Widespread episodic thiamine deficiency in Northern Hemisphere wildlife

https://www.nature.com/articles/srep38821

its likely the pesticides (glyphosate) in the environment, all target Thiamine.

all parasites seem to target Thiamine.

made a liquid concoction of Fenbendazole, B1, Magnesium, Castor Oil & Pine Terpinene.

a little Potassium Citrate to help break down the acids.

just a sip is very powerful.

looks like a pinch of DMSO to B1 will make is pass BBB.

citric acid does help the enzyme activation, but it changes the PH too much, altering the chemistry, might break down the Fenbendazole.

"Thiamine deficiency is ultimately lethal, but it also has a number of preceding sublethal health effects, such as memory and learning disorders, immunosuppression, damage to the blood-brain barrier, neurological disorders, reduced food intake, and altered carbohydrate, protein, and lipid metabolism"

this is all the symptoms of mycoplasma fermentans, glyphosate, chemtrail metals ect..

its all attacking the same thing.

found a few clues, how the Sulfur bond is dependent upon high PH, once it gets to low iit falls apart, and the entire chemistry falls apart, changing color & pungent odors.

Biotoxin
Mycotoxin Deactivation
Enzyme
Nixtamalization (Alkaline)
Esterase
De-Epoxidase

The term "biotoxin" is sometimes used to explicitly confirm the biological origin as opposed to environmental or anthropogenic origins. Biotoxins can be classified by their mechanism of delivery as poisons (passively transferred via ingestion, inhalation, or absorption across the skin).

The latest approach to mycotoxin control is mycotoxin deactivation. By means of enzymes, yeast, or bacterial strains, mycotoxins can be reduced during pre-harvesting contamination. Other removal methods include physical separation, washing, milling, nixtamalization, heat-treatment, radiation, extraction with solvents, and the use of chemical or biological agents.

https://en.m.wikipedia.org/wiki/Mycotoxin

https://en.m.wikipedia.org/wiki/Nixtamalization

https://en.m.wikipedia.org/wiki/Esterase

https://en.m.wikipedia.org/wiki/Defensin

https://en.m.wikipedia.org/wiki/Disulfide

https://en.m.wikipedia.org/wiki/Crotamine

Effects of thiamine on growth, aflatoxin production, and aflr gene expression in A. parasiticus

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5490319/

listening to the conversation tonight.

B1 is Chloroquine Cinchona, its the main component, but may also require a few little parts, but its very close.

B1 was originally discovered as a blue dye, Cinchona Chloroquine is a blue dye.

methionine cycle
holoenzyme complex
neurotropic

B Vitamins in the nervous system: Current knowledge of the biochemical modes of action and synergies of thiamine, pyridoxine, and cobalamin

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6930825/

Prion
Hydrogel

Liquid and Hydrogel Phases of PrPC Linked to Conformation Shifts and Triggered by Alzheimer’s Amyloid-β Oligomers

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6226277/

i dont think it ever occurred to me that Hydrogel is the final product of Prion/Silicon cybernetics.

i was researching these as two different categories, now need to rewire all the ideas back together.

Thiamine is the only compound to directly bind prion (and therefore hydrogel), and my testing is about done, got up to 4000mg a day, now stopping cold turkey, see if the process is healed.

the goal is not to take high dose of B1 forever, just to reset the Prion programming to neutral, to act as an immunity.

Methylene Blue
Thiamine
Quinine
Cytochrome Oxidase

The Effects of Natural and Synthetic Blue Dyes on Human Health: A Review of Current Knowledge and Therapeutic Perspectives

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8634323/

Neurometabolic mechanisms for memory enhancement and neuroprotection of methylene blue

https://www.sciencedirect.com/science/article/abs/pii/S0301008211001948

Reduced activities of thiamine-dependent and cytochrome c oxidase enzymes in cerebral cortex of cattle affected by sulfur-induced polioencephalomalacia

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5644450/

@mikewick77: Candidatus Candidate | Ecency