RE: RE: Protonation Cation/Anion
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RE: Protonation Cation/Anion

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https://en.m.wikipedia.org/wiki/Psychedelic_therapy

https://en.m.wikipedia.org/wiki/Serotonin_receptor_agonist

https://en.m.wikipedia.org/wiki/5-HT2A_receptor

https://en.m.wikipedia.org/wiki/Ibogaine

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Flux (Metallurgy)
Mild Activator
Carboxylic Acid
Acetate

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Acetate
Adenosine
Polyketide
Adenosine A2A Receptor
Microglia

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Adenosine
Adenosine Triphosphate
Adenosine Receptor
Adenosine Diphosphate
Adenosine A2A Receptor
Adenosine Monophosphate

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Enzyme Activator
Acetic
Acetate
Acetylation
Acyl-CoA

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https://en.m.wikipedia.org/wiki/Acetate

https://en.m.wikipedia.org/wiki/Adenosine

/https://en.m.wikipedia.org/wiki/Adenosine_A2A_receptor

https://en.m.wikipedia.org/wiki/Microglia

Acetate supplementation modulates brain adenosine metabolizing enzymes and adenosine A2A receptor levels in rats subjected to neuroinflammation

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4050445/

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well, the mystery is solved, regarding the exact name & the pathways, now we can learn exactly what is happening.

this is slightly convoluted at first look, but its doing a very complicated series of things.

the word antagonist is misleading, because it means blocker, and what i believe we are doing is clearly the pathways Microglia, which then clears brain plaque, healing neurological pathways.

Adenosine A2A Receptor Antagonists in Neurodegenerative Diseases: Huge Potential and Huge Challenges

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5857539/

https://en.m.wikipedia.org/wiki/Adenosine_A2A_receptor_antagonist

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https://en.m.wikipedia.org/wiki/Adenosine

https://en.m.wikipedia.org/wiki/Adenosine_deaminase_deficiency

https://en.m.wikipedia.org/wiki/Adenosine_deaminase_2_deficiency

https://en.m.wikipedia.org/wiki/Tumor_necrosis_factor

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Methylene blue
Methylthioninium chloride

Methyl (Hydrocarbon)
Thioninium (Nitrogen/Sulfur)
Chloride
Glutathione

Acetate
Adenosine
B12
Cobalamin
Methylcobalamin
Corrin
Cobalt