@mikewick77: Boron Molybdenum

the way Fenbendazole binds to parasites, it cleans mitochondria, and with a powerful polarity mineral enzyme, might help increase the effect & detoxification.

yeast seems to move around the body with much more intelligence then bacteria does, morgellens may have more of a yeast then any kind of nano robot nonsense.

all effective medications have a Nitrogen base, and Sulfur always seems to be included in the most powerful variations, and they always exclude the minerals & enzyme connection.

and same with gulf war syndrome & now long covid & vax injured, im convinced only a yeast can be so troublesome, to even infect the gut bacteria or mycoplasma, and mislabeled phage.

my first clue was Craig Venter manufactured Synthia using a yeast in Mycoplasma.

i need to know how they make Suramin & Fenbendazole.

Suramin is from Pine Terpinene, and Fenbendazole is a Polyphenol, Pyrazole & DMSO.

DMSO is critical for the body functions, but taking it while smoking & drinking, flips the polarity the wrong direction, and the DMSO ends up chelating molybdenum, that is required to make the enzymes required for smoke antioxidant & alcohol digestion, then causes a cascade of formaldehyde & cyanide oxides.

so DMSO is only needed as a micronutrient, but the ion polarized minerals is required to flip the oxidants into antioxidant.

opps, found out the hard way on that last year.

but DMSO mixed with liquid smoke, damn that was a really bad idea, it was like smoking 100 packs of cigarettes, so ya.. Phenol & Polyphenol are like oppsite sides of the spectrum regarding health wise.

with the liquid smoke, i was trying to find a way to harvest the organic acids from biomass & trees.

i wasny getting enough feedback from people i was asking about this idea, so the obly way forward was to prove or disprove the idea, so i could then move on to the next phase of ideas, or just drop the idea entirely.

come to find out that the temperature of the fire converts the Carbon into some kind of Oxidative Carbon soot, and has absolutely no medical value whatsoever.

this article explains the Malaria Protozoa Apicoplast idea, because Apicoplast is a Algae closely related to Fungus & Yeast, and Amoebae are Protozoa Invertebrate, similar to insects.

its hard to explain all this stuff, but im sure this is whats making people sick, a symbiogenesis between exotic yeast infecting all kinds of microbes.

The ‘Amoeboid Predator-Fungal Animal Virulence’ Hypothesis

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6463022/

conceptualizing how to manufacture a universal metalloenzyme, to both break up toxic protein, resistant starch, bind to parasitic microbe microtubules, breaking up biofilms, neutralize charged lipids & detoxify.

The role of heat shock proteins in preventing amyloid toxicity

https://www.frontiersin.org/articles/10.3389/fmolb.2022.1045616/full

"The name amyloid stems from this interaction with dyes, which also color starch (amylon). "

the only thing that interacts with amyloid is a very specific dye, thats another clue, bind the dye with a charged metalloenzyme.

Amyloid-Beta
Metallothionein
Transthyretin Binding

Human metallothioneins 2 and 3 differentially affect amyloid-beta binding by transthyretin

https://pubmed.ncbi.nlm.nih.gov/20646067/

Formaldehyde-Crosslinked Nontoxic Aβ Monomers to Form Toxic Aβ Dimers and Aggregates: Pathogenicity and Therapeutic Perspectives

https://chemistry-europe.onlinelibrary.wiley.com/doi/10.1002/cmdc.202100428

Age-related formaldehyde (FA) metabolism disorders lead to the accumulation of FA. In Alzheimer's disease (AD), β-amyloid (Aβ) binding formaldehyde dehydrogenase (FDH) induces its inactivation and FA overload. FA-crosslinked nontoxic Aβ monomers form toxic Aβ dimers, and Aβ-promoted FA generation and FA-elicited Aβ assembly create a vicious cycle.

Biotin

Carboxylase
Holocarboxylase Synthetas
Janus Kinase (JAK)

Biotinylation

https://en.m.wikipedia.org/wiki/Biotinylation

Spike Protein = Amyloid ≈ Starch Resistant, Amylon Starch Dye.

malfunctioning Nitrogen Enzimes make carbohydrates into Protein Spikes?

Alpha-Synuclein
Biotinylation

The Parkinson’s disease protein alpha-synuclein is a modulator of processing bodies and mRNA stability

https://www.sciencedirect.com/science/article/pii/S009286742200592X

Alpha-synuclein (αS) is a conformationally plastic protein that reversibly binds to cellular membranes. It aggregates and is genetically linked to Parkinson's disease (PD). ... Streptavidin pull-down from extracts yielded highly pure and biotinylation-specific αS enrichments

Biotin Ligase
Holocarboxylase Synthetase

https://en.m.wikipedia.org/wiki/Holocarboxylase_synthetase

holocarboxylase synthetase activates other specific enzymes (called biotin-dependent carboxylases) by attaching biotin to them. These carboxylases are involved in many critical cellular functions, including the production and breakdown of proteins, fats, and carbohydrates.

Streptavidin

https://en.m.wikipedia.org/wiki/Streptavidin

Streptavidin can be used as a building block to link biotinylated DNA molecules to create single walled carbon nanotube scaffolds[8] or even complex DNA polyhedra.

my argument is not that B-Vitamins will cure or prevent Amyloid.

just the suspension that Nitrogen & Sulfur ion polarity is being flipped the wrong way & misfolding.

meaning the correct ion polarized minerals will trigger or activate latent enzimes, via metalloenzymes.

reversing the oxidation cascade correctly should also bind to the parasitic host or root cause.

not sure exactly what manufactures enzymes, but my suspension is similar to synthetic blood, hacking via synthetic mitochondria components, so everything appears normal, yet manufacturers metallothionein prions.

is this the next generation gain of function? is this how spike was manufactured?

Machine learning differentiates enzymatic and non-enzymatic metals in proteins

https://www.nature.com/articles/s41467-021-24070-3

Metallothioneins in Prion- and Amyloid-Related Diseases

https://pubmed.ncbi.nlm.nih.gov/26923022/

Xenobiology

https://en.m.wikipedia.org/wiki/Xenobiology

Emerging Paradigms for Synthetic Design of Functional Amyloids

https://www.sciencedirect.com/science/article/abs/pii/S0022283618302468

The roles of gold nanoparticles in the detection of amyloid-β peptide for Alzheimer's disease

https://www.sciencedirect.com/science/article/pii/S2215038221002193

synthetic biology is near limitless in it ability to cause trouble, prion spikes seem to be the biggest problem on the table right now, might as well figure out how it works, and how to unravel it, likely by good metalloenzymes, flip the polarity back to normal.

according to the blueprint Rudolf Steiner layed out 100 years ago, "The Matrix" concept is what he explained as the "Eighth Sphere" manifest as Ahriman.

he says the portion of humanity that enters this artificial reality, will be eventually erased from existence.

i already kind of know what the cure is & how it operates, but its not exactly clear what is going on.

short summary,

Organic Acids:
Hydrogen Carbon Nitrogen Sulphur Oxygen

Mineral Salts:
Calcium Potassium Magnesium Sodium Trace

the Organic Acids & Mineral Salts need to be combined into the opposite polarity as disease operates, both binding & detox via redox process.

binds the parasitic microbes digestion or replication & breaks apart the toxins via enzymes act like a crowbar.

im not convinced that only gold can do this, regular minerals may be capable of being charged in a similar polarity.

the medicine is simple, i know it is, we just need to know exactly what we are looking for, amyloid is the target.

monkey liver endosymbionts.. HIV, hepatitis ect.. bacteria & yeast symbiogenesis.

meaning different species evolve with specific endosymbionts that act as normal cells, but can be cross infected via mycoplasma.

synthetic microbe (cells) dependent upon wireless signal to remain safe, vaxxed shedding toxic to unvaxxed.

this idea came from the original purpose & manufacturing of Synthia (Laboratorium) by Craig Venter.

this synthetic mycoplasma was manufactured using 4 chemicals, yeast & wireless, with the ability to manufacture medication (or poison).

what they do with Synthia Laboratorium is in public domain, but iit takes a little patching together different articles to see the bigger picture.

mycoplasma is a lipid particle carrier, the bacteria itself is not the disease, but the chemical mimicking potential.

in my opinion, the biggest clues are ..

Metallothionein
Formaldehyde

and the ..

Resistant Starch
Starch = Carbohydrate
Carbohydrate = Carbon

im getting a bead on this process.

the only way to unbind Formaldehyde is with Nitrogen, Sulfur & Metalloenzyme reactions, similar to a Redox.

Nitrogen = B Vitamins
Sulfur = DMSO

Metalloenzyme reactions electrolyte metabolism redox may require citric charged minerals, to move along in the correct ion polarity.

antioxidant enzimes

superoxide dismutase
glutathione peroxidise
glutathione reductase
peroxiredoxins cysteine
catalases peroxisomes
lipoic acid (thiol)

Solubility of nicotinic acid in water, ethanol, acetone, diethyl ether, acetonitrile, and dimethyl sulfoxide

https://www.sciencedirect.com/science/article/abs/pii/S0021961411004113

solubility of nicotinic acid is enhanced in DMSO and diminished in the remaining solvents relative to ideal solubility.

Study in Israel Finds Link Between Nitric Oxide

https://healthnews.com/news/study-finds-link-between-nitric-oxide-and-autism/

Nitric Oxide Cycle, Superoxide and Peroxynitrite

https://www.medicalinsider.com/cardiac3.html#nitriccauses

NO is able to perform post-translational modifications in proteins by the S-nitrosylation of the thiol amino acids, which is a physiological mechanism to regulate protein function.

On the other hand, during aging and pathological processes the behavior of NO can turn harmful when reacts with superoxide anion to form peroxynitrite.

Peroxynitrite Mediates Neurotoxicity of Amyloid β-Peptide and Lipopolysaccharide-Activated Microglia

https://www.jneurosci.org/content/22/9/3484

@mikewick77: Boron Molybdenum | Ecency