Artificial Endosymbiosis Symbiogenesis Auxotrophy Alpha-Proteobacteria Bacteriaphage Mitochondria Endosymbiont Symbiont
https://en.m.wikipedia.org/wiki/Auxotrophy
Requiring one or more specific substances for growth and metabolism that the parental organism was able to synthesize on its own. Used with respect to organisms, such as strains of bacteria, algae, or fungi, that can no longer synthesize certain growth factors because of mutational changes.
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Brucella Suis
Neorickettsia Risticii
Detection of the bacterial endosymbiont Neorickettsia in a New Zealand digenean
https://pubmed.ncbi.nlm.nih.gov/27461113/
https://en.m.wikipedia.org/wiki/Neorickettsia_risticii
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Metabolism of the Gram-Positive Bacterial Pathogen Listeria monocytogenes
https://www.ncbi.nlm.nih.gov/labs/pmc/articles/PMC6699642/
Antioxidant and Oxidative Stress: A Mutual Interplay in Age-Related Diseases
https://www.ncbi.nlm.nih.gov/labs/pmc/articles/PMC6204759/
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Auxotrophy
https://en.m.wikipedia.org/wiki/Auxotrophy
Auxotrophic Mutant
https://www.sciencedirect.com/topics/neuroscience/auxotrophic-mutant
https://en.m.wikipedia.org/wiki/Adenosine
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Adenosine is a direct target for Parasitic Auxotrophy, the body requires this for ATP enzime reactions, and some parasites target this metabolism.
minimal genome endosymbionts are called Auxotrophic Mutants.
specifically target the Nitrogen & Sulfur bonds in folding Proteins.
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Ebola-Like Hemorrhagic Fever
https://en.m.wikipedia.org/wiki/Marburg_virus_disease
https://en.m.wikipedia.org/wiki/Alphaproteobacteria
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Engineering yeast endosymbionts as a step toward the evolution of mitochondria
https://www.ncbi.nlm.nih.gov/labs/pmc/articles/PMC6243291/
"engineered E. coli strains to survive in the yeast cytosol and provide ATP to a respiration-deficient yeast mutant. In a reciprocal fashion, yeast provided thiamin to an endosymbiotic E. coli thiamin auxotroph. This readily manipulated chimeric system"
A genome phylogeny for mitochondria among alpha-proteobacteria and a predominantly eubacterial ancestry of yeast nuclear genes
https://pubmed.ncbi.nlm.nih.gov/15155797/
Effects of mycoplasma contamination on phenotypic expression of mitochondrial mutants in human cells
https://pubmed.ncbi.nlm.nih.gov/6965101/
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the target of cancer & virus are mitochondria.
mitochondria replacement, hostile takeover, mimicking mRNA into DNA.
zombie mitochondria
the way mycoplasma will mimic chemicals to induce a chemotherapy like reaction.
chemicals that reduce white blood cell formation.
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Mito is our power house
Pathogens hijack them
Then our body will look for anaerobic respiration for energy.
https://en.m.wikipedia.org/wiki/Anaerobic_respiration
Anaerobic respiration is respiration using electron acceptors other than molecular oxygen (O2). Although oxygen is not the final electron acceptor, the process still uses a respiratory electron transport chain.
Sulfur & Nitrogen are targets for mycoplasmas, disrupting the natrual formation of enzime & protein folding, resulting in the production of Hydrogen Peroxide.
mycoplasma is most effective in a low Nitrogen & Sulfur environment, so to disrupt the mitochondria process, even though Mycoplasma requires the Nitrogen Sulfur, this is the only way to bind them, the weak point is identical to the process being exploited.
Kidney is only second to the heart in mitochondrial count and oxygen consumption
if cancer could be enhanced to a fast progression, would the symptoms be identical to Ebola?
MYCOPLASMAS AND CANCER: FOCUS ON NUCLEOSIDE METABOLISM
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DMSO Represses Inflammatory Cytokine Production from Human Blood Cells and Reduces Autoimmune Arthritis
https://www.ncbi.nlm.nih.gov/labs/pmc/articles/PMC4816398/
Artificial Mitochondria
Nanobioreactors
Programmed exosome fusion for energy generation in living cells
https://www.nature.com/articles/s41929-021-00669-z
https://en.m.wikipedia.org/wiki/Catechol
https://en.m.wikipedia.org/wiki/Sulfonic_acid
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"But thanks to the system’s plug-and-play mode—just pop in your favorite chemical reactants—we now have a new way to control and reprogram a living cell’s life."
i want to know how similar mRNA & Gain of Function is.
both mRNA vaccines & Gain of Function use Fetus Stem Cell & Mouse DNA?
both are transferring mouse Mitochondria Endosymbionts into humans.
RE: Disease-X